Patients with bronchiectasis: look for specific causes.

manifestations have been previously reported, though not well-documented, and usually with coronary artery aneurysm. Our HHT patient had a well-documented myocardial infarction with normal coronary arteries, as shown by our biology data, ECG, TTE, coronarography and RMI results. The ischaemic myocardial lesions might result from our patient’s inherited thrombophilia: factor V Leiden and MTHFR. In 2002, a case of recurrent ischaemic stroke was reported by Undas et al. in a patient with HHT and factor V Leiden antiphospholipid antibodies. However, this association between inherited thrombophilia and myocardial infarction remains the subject of debate. Rare cases of coronary artery aneurysms without stenosis have been previously reported. With angiographically normal arteries, we hypothesize that in our patient, HHT may be complicated by an endothelial dysfunction. This mechanism of endothelial dysfunction should be verified by an acetylcholine test. Nevertheless, in our opinion, inherited thrombophilia and oral contraceptive therapy have probably contributed to the myocardial ischaemic lesions in our patient.